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Tren experience?


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On paper it rates as 5 times more anabolic and five times more androgenic than testosterone! It does not aromatise to estrogen so no estrogen control is required. You keep a very high percentage of you gains from Tren and it has the ability to permanently deactivate some cortisol receptors! It is great when combined with EQ for a slight fluid buffer for the joints but should not be combined with deca as the are both derivatives of nandrolone and may cause some unwanted side effects. tren doses should be kept at under 500mg/week for even the advanced bodybuilders as high doses can be harsh on the liver and kidneys :)

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Love tren.

If you have not used tren before then start off on some tren acetate rather than tren enanthate, the reason being if you do get any side effects that do not agree with you then the acetate will be out of your system quickly rather than having to put up with a week or so of sides if using enanthate.

There is a range of possible sides including progesterone gyno, not to be confused with estrogen related gyno from test. There are meds that you can take to prevent this.

The only sides I get is my body temp goes up and I get some night sweats, not as bad as a mate of mine though who wakes up every morning with the entire bed soaked, don't know how his partner puts up with it, maybe she likes it. :lol:

Currently on a 180mg tri tren blend consisting of 60 mg tren acetate, 60mg tren enanthate and 60mg tren hexa benzhydra caproate. I am pinning this every second day.

Pretty hard to beat tren if you want some good solid muscle growth. If I only had the choice to use two types of gear then it would be a test/tren combo every time.

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TWL was that PRL induced? i remember reading you ran some bromo, did that take care of it or did you have to run letro?

clash just so you know, im running tren right now :P im just interested in the boards use of it.

chem i suggest you dont interest yourself in something like tren without having cycled before.

hone that tri tren is suppose to be gud stuff, expensive too :pfft:

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All my experiences with Tren have been positive. The Strength gains are phenomenal. Post cycle mass retention is also great. Some hormonal sweets but no progesterone related gyno for me. Slight Tren cough at doses over 400mg/week. Favorite is Tren Acetate. I agree completely with "HONE" my pick of two all time favorite AAS would be Test & Tren for the andrgenic dominance period of a mass phase and tren and EQ for and anabolic dominance period. Tren can swing both ways. I use it at least 6months out of every year and especially when doing a course of peptides. :nod:

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Is there much of a difference between normal estrogen gyno and progesterone gyno? ie, is it still the physical development of breast tissue, or is there some other cause?

Clash - your profile says you're female, and yet you're using particularly potent androgenic AAS. Don't take this the wrong way, but did you tick the right gender when you registered? :)

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Is there much of a difference between normal estrogen gyno and progesterone gyno? ie, is it still the physical development of breast tissue, or is there some other cause?

Clash - your profile says you're female, and yet you're using particularly potent androgenic AAS. Don't take this the wrong way, but did you tick the right gender when you registered? :)

Must have been my feminine side showing through. A wee bit estrogen dominant when I registered. :D . Never took much notice of that. Thanks for the heads up. Have taken an extra 100mg of Proviron and my gender miraculously changed :doh:

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ive been running tren a for the last 10 days @ 50mg ED.

so far so good. getting most of the sides, good and bad. tren has made breathing quite a challenge lol. its been a while since ive done any cardio so i thought id treat myself this evening after my workout. im left wondering if this is what a chain smoker feels like :lol:

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ive been running tren a for the last 10 days @ 50mg ED.

so far so good. getting most of the sides, good and bad. tren has made breathing quite a challenge lol. its been a while since ive done any cardio so i thought id treat myself this evening after my workout. im left wondering if this is what a chain smoker feels like :lol:

How many cycles have you run before this one?

This is your first tren cycle I take it?

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ive been running tren a for the last 10 days @ 50mg ED.

so far so good. getting most of the sides, good and bad. tren has made breathing quite a challenge lol. its been a while since ive done any cardio so i thought id treat myself this evening after my workout. im left wondering if this is what a chain smoker feels like :lol:

How many cycles have you run before this one?

This is your first tren cycle I take it?

yeah, my first tren cycle

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ive been running tren a for the last 10 days @ 50mg ED.

so far so good. getting most of the sides, good and bad. tren has made breathing quite a challenge lol. its been a while since ive done any cardio so i thought id treat myself this evening after my workout. im left wondering if this is what a chain smoker feels like :lol:

How many cycles have you run before this one?

This is your first tren cycle I take it?

yeah, my first tren cycle

How many cycles have you done in total?

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There is nothing to combat progesterone gyno/sides unfortunately.

Caber/Bromo/Prami all target Prolactin.

I understand that the general school of thought is that by reducing prolactin progesterone becomes less effective.

Some may disagree.

Androgens decrease prolactin. Progestins like Tren & Deca bind at the Progesterone Receptor & high levels of progesterone can raise the levels of prolactin. Prolactin is the culprit that causes increased breast tissue growth, libido & ED & lactation in extreme cases. Prog Gyno cannot develop though in the absence of Estrogen so its important to control Estrogen /Test+DHT ratio when running Tren although it does not convert to E it does to Prog.

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There is nothing to combat progesterone gyno/sides unfortunately.

Caber/Bromo/Prami all target Prolactin.

I understand that the general school of thought is that by reducing prolactin progesterone becomes less effective.

Some may disagree.

Androgens decrease prolactin. Progestins like Tren & Deca bind at the Progesterone Receptor & high levels of progesterone can raise the levels of prolactin. Prolactin is the culprit that causes increased breast tissue growth, libido & ED & lactation in extreme cases. Prog Gyno cannot develop though in the absence of Estrogen so its important to control Estrogen /Test+DHT ratio when running Tren although it does not convert to E it does to Prog.

from my understanding dopamine secreted by the hypothalamus inhibits production of prolactin from the pituitary. dopamine agonists activate the dopamine receptor in absence of dopamine or in our case elevated PRL. can you explain how upregulated progesterone receptors stimulate this production?

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There is nothing to combat progesterone gyno/sides unfortunately.

Caber/Bromo/Prami all target Prolactin.

I understand that the general school of thought is that by reducing prolactin progesterone becomes less effective.

Some may disagree.

Androgens decrease prolactin. Progestins like Tren & Deca bind at the Progesterone Receptor & high levels of progesterone can raise the levels of prolactin. Prolactin is the culprit that causes increased breast tissue growth, libido & ED & lactation in extreme cases. Prog Gyno cannot develop though in the absence of Estrogen so its important to control Estrogen /Test+DHT ratio when running Tren although it does not convert to E it does to Prog.

from my understanding dopamine secreted by the hypothalamus inhibits production of prolactin from the pituitary. dopamine antagonists activate the dopamine receptor in absence of dopamine or in our case elevated PRL. can you explain how upregulated progesterone receptors stimulate this production?

There is not a lot of information on this & as I'm not an endocrinologist I cant say but from what I have read prolactin secretion seems to be under the control of the dopaminergic neuronal system, so it is thought that somehow progesterone interacts with this system to increase prolactin. Dopaminergic agonists like Caber and Bromo inhibit prolactin secretion, whereas a large number of drugs, including antidepressants and antipsychotics, stimulate prolactin secretion. this is often the cause of libido & ED issues in AD users & drug addicts like meth where there is an initial sexual high followed by decreasing libido as the drug becomes established more permanently in the body.

Also anabolic steroids seem increase the metabolic breakdown of both dopamine and serotonin in the human body. Low serotonin levels have been implicated in anxiety, aggression, violent behavior, and impulsiveness, as well as depression. This may be a the cause of "roid rage". The decrease in dopamine may very well be responsible for an increase in prolactin in steroid users. This, coupled with excess estrogen from aromatizable androgens could be a cause of gynecomastia.

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Androgens decrease prolactin. Progestins like Tren & Deca bind at the Progesterone Receptor & high levels of progesterone can raise the levels of prolactin. Prolactin is the culprit that causes increased breast tissue growth, libido & ED & lactation in extreme cases. Prog Gyno cannot develop though in the absence of Estrogen so its important to control Estrogen /Test+DHT ratio when running Tren although it does not convert to E it does to Prog.

from my understanding dopamine secreted by the hypothalamus inhibits production of prolactin from the pituitary. dopamine antagonists activate the dopamine receptor in absence of dopamine or in our case elevated PRL. can you explain how upregulated progesterone receptors stimulate this production?

There is not a lot of information on this & as I'm not an endocrinologist I cant say but from what I have read prolactin secretion seems to be under the control of the dopaminergic neuronal system, so it is thought that somehow progesterone interacts with this system to increase prolactin. Dopaminergic agonists like Caber and Bromo inhibit prolactin secretion, whereas a large number of drugs, including antidepressants and antipsychotics, stimulate prolactin secretion. this is often the cause of libido & ED issues in AD users & drug addicts like meth where there is an initial sexual high followed by decreasing libido as the drug becomes established more permanently in the body.

Also anabolic steroids seem increase the metabolic breakdown of both dopamine and serotonin in the human body. Low serotonin levels have been implicated in anxiety, aggression, violent behavior, and impulsiveness, as well as depression. This may be a the cause of "roid rage". The decrease in dopamine may very well be responsible for an increase in prolactin in steroid users. This, coupled with excess estrogen from aromatizable androgens could be a cause of gynecomastia.

is there anything that can metabolize progesterone apart from letro?

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from my understanding dopamine secreted by the hypothalamus inhibits production of prolactin from the pituitary. dopamine antagonists activate the dopamine receptor in absence of dopamine or in our case elevated PRL. can you explain how upregulated progesterone receptors stimulate this production?

There is not a lot of information on this & as I'm not an endocrinologist I cant say but from what I have read prolactin secretion seems to be under the control of the dopaminergic neuronal system, so it is thought that somehow progesterone interacts with this system to increase prolactin. Dopaminergic agonists like Caber and Bromo inhibit prolactin secretion, whereas a large number of drugs, including antidepressants and antipsychotics, stimulate prolactin secretion. this is often the cause of libido & ED issues in AD users & drug addicts like meth where there is an initial sexual high followed by decreasing libido as the drug becomes established more permanently in the body.

Also anabolic steroids seem increase the metabolic breakdown of both dopamine and serotonin in the human body. Low serotonin levels have been implicated in anxiety, aggression, violent behavior, and impulsiveness, as well as depression. This may be a the cause of "roid rage". The decrease in dopamine may very well be responsible for an increase in prolactin in steroid users. This, coupled with excess estrogen from aromatizable androgens could be a cause of gynecomastia.

is there anything that can metabolize progesterone apart from letro?

You mean inhibit progesterone. Im not too sure. Good question. There is some links I recall about Hcg & Progesterone. Hcg is used to stimulate the placenta in pregnant women to bring on birth. I'll have to check. Typically you control estrogen not progesterone which is really only a problem if using progestins like Tren.

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