Steroid-related case report I just came across

[Diab0lic]

Rothman RD, Weiner RB, Pope H, Kanayama G, Hutter Jr AM, Fifer MA, Dec Jr GW, Baggish AL. Anabolic androgenic steroid induced myocardial toxicity: An evolving problem in an ageing population. BMJ Case Reports : [3 pages], Aug 2011.

A 62-year-old man developed myocardial toxicity during use of metandienone and nandrolone to increase muscle mass and strength.

One year before presentation, the man started injecting IM nandrolone 400−800 mg/week and metandienone 20−40 mg/week. After about 6 months of use he started to experience decreased exercise stamina with progressive shortness of breath. On presentation, examinations revealed sinus bradycardia with a diffuse and left laterally displaced point of maximal impulse, jugular venous distension, a soft left-sided S3 gallop, bibasilar crackles and mild hepatomegaly. ECG showed left ventricle dilation with impaired left ventricular systolic function, diffuse hypokinesis and diastolic dysfunction. He was diagnosed with non-ischaemic cardiomyopathy.

The man was recommended abstinence from anabolic androgenic steroids and started receiving a heart failure regimen including lisinopril and furosemide. At 12-month follow-up, his left ventricle size and function had normalised, and his exercise tolerance had increased. Several years after discontinuing the anabolic androgenic steroids, he was continuing to train without limitations.

\:D/

[severe]

Interesting, but it needs to represent a sizeable number of patients to be a global concern. One man is not enough.

and where was the testosterone? What was the purpose of the aas used, in the abscence of test? Sounds like they set it up to fail.

[Grover]

and he was 62 which is pretty damn good to be still lifting!