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Anavar


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Just wanted to ask your experiences with var were?

I've heard mixed reviews no doubt from various sources and qualities. I've always wanted to try it, in fact it was my first option before injectables, I quickly got talked out of that but Var still captures my attention everytime it comes up. I planed to run it alone 50-80 mg a day. Like a bridge.

Sounds like Var with an AI gave some good results, surely not sustainable but worth it?

at around 85kg LBM would 50-80mg a day be sufficient?

I have just finished my first cycle of just test e, reviewing what I have done, what I could've done better and whats next for me. Certainly a new way of life.

Peace

-C

[MOD EDIT: Split from Sust + Winny topic.]

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Anavar has always been a favorite of mine when it has been available. Whether it is for bridging, mass phase of pre-comp it is misunderstood and often overlooked for many reasons including price.

On paper - milligram for milligram it displays as much as 6 times the anabolic effect of testosterone with significantly less androgenicity. it has no significant estrogenic or progestational activity and has very little impact on the HPTA axis even at moderate to high doses.

Although all AAS promote protein synthesis, Oxandrolone (Anavar) is unmatched in this area.

Its tendency is to promote pure lean mass gains with no fat or water.

Most AAS users do not actually feel the effects of Anavar as other AAS being used at the same time tend to drown it out. But this does not mean it is not working, and it is because it is not "felt" that has led some people to believe it is not worth the money.

I have taken Anavar on its own after an extensive break from AAS and the pump and strength increase were very impressive. I have also taken it during mass and pre-comp phases and have attributed Anavar to many of my successes on stage

I rate Anavar 8/10 and if it was within the budget, I would recommend Anavar for all phases for men and woman.

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Oxandrolone was designed to be safe for use by women and children. Although all AAS supress endogenous testosterone production to some degree, Oxandrolone is no exception but it is the degree of suppression that makes the impact of Anavar mild in comparison. One study cited a 45% reduction in serum testosterone levels at doses of 40mg/day after 12 weeks and at 80mg per day the reduction was 66%. Compare this to just 8 weeks of 15mg of Dbol causing a reduction of 70% and you can see how it can be concluded that Anavar has comparatively little impact on HPTA axis.

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Oxandrolone was designed to be safe for use by women and children. Although all AAS supress endogenous testosterone production to some degree, Oxandrolone is no exception but it is the degree of suppression that makes the impact of Anavar mild in comparison. One study cited a 45% reduction in serum testosterone levels at doses of 40mg/day after 12 weeks and at 80mg per day the reduction was 66%. Compare this to just 8 weeks of 15mg of Dbol causing a reduction of 70% and you can see how it can be concluded that Anavar has comparatively little impact on HPTA axis.

can i take a look at this study?

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Search medical studies of Oxandrolone on HIV+ men. I have only saved the txt not the link but can find it if you are unable to track it down...basically - The study looked at escalating doses (20mg, 40mg, 80mg) of Oxandrolone in 262 HIV+ men. The study showed conclusively that Oxandrolone was measurably safer in Hepatotoxicity than other alkylated agents and had less impact on HPTA axis :)

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Search medical studies of Oxandrolone on HIV+ men. I have only saved the txt not the link but can find it if you are unable to track it down...basically - The study looked at escalating doses (20mg, 40mg, 80mg) of Oxandrolone in 262 HIV+ men. The study showed conclusively that Oxandrolone was measurably safer in Hepatotoxicity than other alkylated agents and had less impact on HPTA axis :)

there are hundreds of articles on oxandrolone's medical use and you expect me to look for it after i ask you to provide this evidence? below is an article that illustrates oxandrolones suppressive nature.

Effect of low dose oxandrolone and testosterone treatment on the pituitary-testicular and GH axes in boys with constitutional delay of growth and puberty.

Crowne EC, Wallace WH, Moore C, Mitchell R, Robertson WH, Holly JM, Shalet SM.

Department of Endocrinology, Christie Hospital Trust, Manchester, UK.

OBJECTIVE: To investigate the effect of low dose oxandrolone and testosterone on the pituitary-testicular and GH-IGF-I axes. DESIGN: Prospective double-blind placebo-controlled trial. PATIENTS: Sixteen boys with constitutional delay of growth and puberty (CDGP) with testicular volumes 4-6 ml were randomized to 3 months treatment: Group 1 (n = 5), daily placebo: Group 2 (n = 5), 2.5 mg oxandrolone daily or Group 3 (n = 6), 50 mg testosterone monthly intramuscular injections with assessment (growth, pubertal development and overnight hormone profiles) at 0, 3, 6 and 12 months. MAIN OUTCOME MEASURES: lh - leutenizing hormone - and GH profiles (15-minute samples) were analysed by peak detection (Pulsar), Fourier transformation and autocorrelation. testosterone levels were measured hourly and insulin, sex hormone binding globulin , IGF-I, and IGFBP-3 levels at 0800 h. Statistical analysis was by multivariate analysis of variance for repeated measures. RESULTS: lh - leutenizing hormone - and testosterone parameters increased significantly with time in all 16 (lh - leutenizing hormone - AUC, P < 0.001; peak amplitude, P = 0.02; number of peaks, P = 0.02; testosterone AUC, P = 0.02; morning testosterone, P = 0.002). In Group 2, however, lh - leutenizing hormone - and testosterone parameters decreased at 3 months followed by a rebound increase at 6 and 12 months. sex hormone binding globulin levels were markedly reduced at 3 months (P = 0.006) and a wider range of dominant GH frequencies was present although GH AUC was not increased until 6 months, with an increase in GH pulse frequency but not amplitude. IGF-I levels were increased at both 3 and 12 months. In Group 3, pituitary-testicular suppression was not apparent, but GH levels increased with an increase in GH amplitude at 3 and 12 months. CONCLUSION: Oxandrolone transiently suppressed the pituitary-testicular axis and altered GH pulsatility. testosterone increased GH via amplitude modulation.

chem next time get the mod to delete both of your posts.

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Search medical studies of Oxandrolone on HIV+ men. I have only saved the txt not the link but can find it if you are unable to track it down...basically - The study looked at escalating doses (20mg, 40mg, 80mg) of Oxandrolone in 262 HIV+ men. The study showed conclusively that Oxandrolone was measurably safer in Hepatotoxicity than other alkylated agents and had less impact on HPTA axis :)

there are hundreds of articles on oxandrolone's medical use and you expect me to look for it after i ask you to provide this evidence? below is an article that illustrates oxandrolones suppressive nature.

Effect of low dose oxandrolone and testosterone treatment on the pituitary-testicular and GH axes in boys with constitutional delay of growth and puberty.

Crowne EC, Wallace WH, Moore C, Mitchell R, Robertson WH, Holly JM, Shalet SM.

Department of Endocrinology, Christie Hospital Trust, Manchester, UK.

OBJECTIVE: To investigate the effect of low dose oxandrolone and testosterone on the pituitary-testicular and GH-IGF-I axes. DESIGN: Prospective double-blind placebo-controlled trial. PATIENTS: Sixteen boys with constitutional delay of growth and puberty (CDGP) with testicular volumes 4-6 ml were randomized to 3 months treatment: Group 1 (n = 5), daily placebo: Group 2 (n = 5), 2.5 mg oxandrolone daily or Group 3 (n = 6), 50 mg testosterone monthly intramuscular injections with assessment (growth, pubertal development and overnight hormone profiles) at 0, 3, 6 and 12 months. MAIN OUTCOME MEASURES: lh - leutenizing hormone - and GH profiles (15-minute samples) were analysed by peak detection (Pulsar), Fourier transformation and autocorrelation. testosterone levels were measured hourly and insulin, sex hormone binding globulin , IGF-I, and IGFBP-3 levels at 0800 h. Statistical analysis was by multivariate analysis of variance for repeated measures. RESULTS: lh - leutenizing hormone - and testosterone parameters increased significantly with time in all 16 (lh - leutenizing hormone - AUC, P < 0.001; peak amplitude, P = 0.02; number of peaks, P = 0.02; testosterone AUC, P = 0.02; morning testosterone, P = 0.002). In Group 2, however, lh - leutenizing hormone - and testosterone parameters decreased at 3 months followed by a rebound increase at 6 and 12 months. sex hormone binding globulin levels were markedly reduced at 3 months (P = 0.006) and a wider range of dominant GH frequencies was present although GH AUC was not increased until 6 months, with an increase in GH pulse frequency but not amplitude. IGF-I levels were increased at both 3 and 12 months. In Group 3, pituitary-testicular suppression was not apparent, but GH levels increased with an increase in GH amplitude at 3 and 12 months. CONCLUSION: Oxandrolone transiently suppressed the pituitary-testicular axis and altered GH pulsatility. testosterone increased GH via amplitude modulation.

chem next time get the mod to delete both of your posts.

Yes unfortunately I do expect you to find the research for yourself. Try Martorana G, Concetti S, Manferrari F, Creti S. J Urol. 1999 Dec; 162(6):2089

What I posted was not opinionated, it was from medical literature and can be found without too much difficulty.

I wil not copy and paste the study on the forum though because the technical information is only sort by the minority and the basic information in layman's terms is mostly what people are looking for so they can digest and clearly understand the information and then apply it to their own personal situation to help them make an informed decision or choice. :)

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From what I can find you can purchase the articale from "Science Direct" but you can find refernces taken from it at:

http://xsteroids.com/drug-profiles/anab ... androlone/

http://www.anavarshop.com/Steroid-Artic ... fects.html

http://www.building-body.com/forum/view ... =17&t=4068

But this is just all copy and pasted repition from the original source material and not the study itself.

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