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Daz69 last won the day on June 28

Daz69 had the most liked content!


About Daz69

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    Senior member
  • Birthday July 5

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    Dead for tax purposes...
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    Training injured....
  • Steroid use

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  1. hMG is now preferred as it is a combo of LH and FSH... Some endo's include Clomiphene also..
  2. UG sustanon 350

    Isn't hCG a LH mimicking agent..? Estradiol will eventually clear via it's metabolic pathway, only slower than testosterone, thats why its prudent to taper off end of cycle and employ low dose AI to avoid estrogen dominance..

    Depends on the dose, strength increase, some extra aggression.. No you won't need PCT, it's pointless anyway..
  4. Doctors Supervision


    That's all we had when I started.. Ok if you can obtain genuine product..
  6. Test cycle

    I personally try to reduce harms from AAS on-cycle (via use of antioxidants Taurine 3g - Royal Jelly 1g), and I use incredibly small doses compared to what's typically promoted online, just enough to push me into upper-normal and slightly-supraphysiologic levels over the duration of a progressively up-tapering cycle. I would use ten times that amount in the past with no noticeable improvement in long-term gains or progress. The use of SERMs is no guarantee of recovery whatsoever. And guys kid themselves if they think using HCG to maintain testes size on cycle is going to protect them from harmful effects on-cycle - it does not... Generally speaking LH/FSH come back within days of the end of a cycle, making most PCT drugs utterly pointless. The issue seems to be the failure of the message (LH/FSH) to get through to the testes, possibly because of massive leydig cell death... Although some people are naturally less affected, probably because they have higher innate levels of antioxidants like glutathione... A recent study highlights just how hard it is for former AAS users to recover natural testosterone levels after they finish using steroids. To be clear, this doesn't really elucidate which compounds are easier or harder on the HPTA - we already know nandrolone is spectacularly hard to recover from, but at least 8 compounds were used by more than 50% of the cohort - just that as a group it's unlikely we'll ever recover to pre-steroid levels or to those comparable with age-matched non-users. So what help is the study? Well it does give us three things to consider: (1) prospective new users of AAS should always be made aware that recovery is not a foregone conclusion, and that they may never recover former levels of testosterone (2) most PCTs are a complete waste of time and money in the long-run, even if they may temporarily boost test levels (3) taking as many precautions as possible to protect the HPTA (and especially testes) from free-radical damage should be prioritised on cycle (eg. controlling aromatase/oestrogen levels, using supplements like curcumin, royal jelly and taurine). https://journals.plos.org/plosone/article?id=10.1371/journal.pone.0161208
  7. Test cycle

    Obviously a larger dose should yield greater effect, but has likelyhood to induce greater side effects.. That is basic pharmacodynamics there is plenty information out there to show this statement is correct.. Given your height and current bodyweight 250mg/week (possibly split into 2 doses of 125mg ) might better suit your personal physiology.. You shouldn't require PCT as in my opinion it's a load of bollocks, taking care of the testes on-cycle to avoid damage via the supplementation of antioxidants, might seem more prudent, as opposed to trying to restart testicular function once sertoli and leydig cells are dead or damaged.. Train hard and eat well...
  8. Cheat Meals/Days

    Why do you think a high calorie day should consist of shite, ever thought of increasing good fat consumption instead of processed garbage and refined sugar known to cause serious pathophysiological conditions..
  9. Half-life of enanthate is just over 4 days, so not what you read on most popular steroid boards.. Should peak blood plasma levels by about 20 days, but kicks in at roughly 2 hours after first injection..
  10. Remember I've been involved for the last 38 years, that's how it used to be back home in the 70's - 90's, 3 months on hormones, 3 months off on SERM's... Until blast cruise methodology surfaced..
  11. The argument is that you could just have recovered fine without PCT, but by taking it you believe it worked.!!
  12. To PCT or not to PCT: 1) typical recovery issues from use of AAS- primary hypogonadism (hypergonadotrophic hypogonadism) ---> LH/FSH = high, test = low, sperm quality = low- secondary hypogonadism (hypogonadotrophic hypogonadism) ---> LH/FSH = low, test = low, sperm quantity = low- partially (or fully) both forms ---> probably typical for majority of guys to lesser or greater degree2) typical traditional PCTs recommended online- hcg (during, after)- SERMS (after)- aromatase inhibitor (lower background oestrogen)3) standard theory of PCTs- maintain testes size with LH mimetic (hcg) = 'protective' (but how? what proof?)- greatly elevated LH = faster gonad recovery (yet natural LH recovers rapidly - how high is healthy and is it really 'faster'?)- hCG 'sensitises' testes for restored natural LH pulse (is there actually any proof for this assertion?)- lowering oestrogen = reduce negative feedback (oestrogen longer half-life = high relative levels post-cycle)4) population studies and case studies showing failure to recover- several already posted and discussed, summarise/copy-paste- case studies (eg https://www.ncbi.nlm.nih.gov/pubmed/21575947; https://www.ncbi.nlm.nih.gov/pubmed/21682835)- HCG maintains size, does not necessarily increase testosterone (ie leydig cell number)(via here)- hcg fails completely (https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1022657)- hcg may hinder long-term recovery (https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2360778)- SERM-induced LH increase = temporary bandaid - transient and not actual 'recovery'- most SERM and hCG studies performed on NORMAL people with hypogonadism not AAS users who have damaged HPG axis cycling supraphysiological levels of hormones5) probable reasons why treatments fail- negative feedback probably not the cause of most chronic recovery failures - therefore treatments designed to treat that are missing the point- LH/FSH recover very quickly post-cycle anyway- hCG long half-life = bleed; natural GnRH, LH/FSH = pulsatile- damage to testes from AAS or metabolites/oestrogen- damage to pituitary from AAS or metabolites/oestrogen- damage to hypothalamus from AAS or metabolites/oestrogen6) possible mechanisms to limit damage - low dose cycling/tapering (law of diminishing returns)- aromatase inhibitor with potently aromatising compounds to control damaging estradiol metabolites- reduce reliance on potentially more harmful compounds (eg dbol = more potent and harmful metabolic oestrogens; deca stays in system for months, etc)- utilise shorter duration cycles- avoid blast n cruise unless competing/great genetics - be patient! bodybuilding = lifestyle not race.- supplements to attenuate oxidative harms on cycle (various studies in animals = best we've got; theory = sound)- potential for triptorelin in secondary HG, possibly others like lowering prolactin (indicates injured hypothalamus) with pramipexole etc. May not 'heal' damage caused on cycle though, any more than clomid does.7) take home messages/conclusions - SERMs & HCG may help some feel better but others worse, unlikely to promote long-term recovery - moreso bandaid- therefore PCT is no guarantee of recovery and must be presented and admitted as such (too much convenient dishonesty)- however, feel free to try PCT and use blood tests but to repeat also:- (a) consider lower dosed cycles and realistic maintainable gains- (b) consider use of supplements to potentially attenuate harms
  13. Any method that provides minimal oxidative stress throughout the cycle, should make successful recovery possible, either by supplementation on cycle and or very low dose AI on cycle, or as you taper off.. 100mg (which in reality is only 65mg of actual hormone) in the last 2 weeks then off, should work just fine, as blood testosterone will lower, as residual test already in blood half lifes over the next couple of weeks.. Nolva clomid only gained popularity on some steroid boards due to dealers needing to make a profit out of time off, which was the old school methodology ie: 3 months on 3 months off.. No amount of mimicking LH, or stimulating GnRH will work over the longer term if sertoli or leydig cells are dead, or present in significantly reduced number..
  14. Also remember a mobile phone in your pocket can cause similar damage to leydig and sertoli cells via low frequency EMF exposure activating voltage gated calcium channels, creating reactive oxygen and also more dangerous reactive nitrogens..
  15. As previously stated estrogen on cycle can be a very personal thing dependant upon a plethora of factors, although remember some estrogen is needed for growth, unfortunately this can leave you exposed to issues with free radicals. Think of aromatase as a polluting, free-radical generating 'factory', taking in testosterone, hydroxylating it multiple times and oxidating off methyl groups as carboxylic acids (eg formate), in the production of oestrogens/oestradiol. If concentrations of aromatase and testosterone rise in tandem beyond physiological norms, the stage is set for free-radical generation that can rapidly overload innate anti-oxidative mechanisms (GSH etc), causing apoptosis of neighbouring cells and ultimately inflammation that can then aggravate the whole situation in a vicious positive-feedback cycle. Also bear in mind that oestrogens churned out and subsequently metabolised can themselves become problematic eg catechol oestrogens, while even 17-B-oestradiol has been shown to be innately toxic, genotoxic and carcinogenic in certain situations/organs, largely via free-radical generating behaviour/activation. However, since most men suffer from longer-term effects and a failure to fully recover after various cycles of AAS, some worse than others, this is generally regarded as proof that the AAS themselves - rather than merely the decline in LH/FSH levels as a result of that negative feedback and shrinkage of the testes - cause long-term harm to cells in the testes, and probably the hypothalamus and pituitary as well. And there is weak evidence for transiently elevated LH/FSH from SERMs undoing that damage. Most will enjoy a temporary boost to test levels, only to see them decline back to a lower baseline once off. In fact, you could speculate their use and the transient boost to test may actually hamper underlying recovery, since most SERMs diminish growth hormone levels, and there is a mechanism via elevated GH and local IGF-1 for the recovery of leydig cell number and density in testes. Some of the known harms from AAS cycles include direct oxidative damage to various cells in the testes and brain. And there is some experimental evidence that various free radical scavengers can attenuate these oxidative harms or balance natural recuperative systems. So in terms of cost/benefit it's likely very worthwhile to take some additional supplements on cycle that may help, like taurine, NAC, astragalus, raw cacao, royal jelly, bioavailable curcumin, etc. Whether you wish to approach the oxidative issue via an AI as well as supplementation is up to you, but there is evidence that combinations of the above supplements do work..